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resulting in superoxide, a byproduct characteristic of causing cellular damage . However, sodium azide, superoxide dismutase, and catalase have been seen to inhibit the autoxidation of myricetin.

Myricetin may also act as a pro-oxidant in its ability to increase the production of hydroxy radicals through reactions with Fe²⁺ or Fe³⁺-EDTA and hydrogen peroxide.

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