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These mutants presented excessive growth in the axillary buds, which induced their terminal stem to start branching abnormally. Previously, cytokinins were thought to be the only molecule involved in the regulation of stem branching, but these mutants presented normal production and signaling of cytokinins, leading to the conclusion that another substance was acting on the axillary buds. Different tests that consisted in inserting part of the mutants plants into wild specimens

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